Day 1 :
- Neonatology & Perinatology
Location: Japan
Session Introduction
Dita Indriyati
Dita Indriyati, Anik Rustiyaningsih, Susetyowati Faculty of Medicine, Public Health, and Nursing Universitas Gadjah Mada
Title: The Difference of Binge Eating Disorder Tendencies on Adolescents with High and Normal Nutritional Status in Yogyakarta
Time : 9:00 AM
Biography:
Dita Indriyati has completed her bachelor’s degree in nursing at the age of 22 years from Universitas Gadjah Mada and now she is a student of professional education programs (nurses) in Faculty of Medicine, Public Health, and Nursing Universitas Gadjah Mada.
Abstract:
Background: Binge eating disorder is a type of eating disorder that is commonly found in adolescents with high nutritional status. However, in a larger population, binge eating disorder can also be experienced by adolescents with normal nutritional status. By the time this research was conducted, there was still no research that compared about binge eating disorder on both adolescents with high and normal nutritional status. Objective: To compare binge eating disorder tendencies on both adolescents with high and normal nutritional status in Yogyakarta. Method: Quantitative observational approach was applied by using a cross-sectional study involving 454 adolescents aged 13-15 years old. Data of the nutritional status were analyzed by using BMI-for-age, while the tendencies level of binge eating disorder was measured by using The Adolescent Binge Eating Disorder questionnaires. Results: Based on the chi-square analysis, binge eating disorder tendencies status on 454 adolescents with high and normal nutritional status is significantly different (p=<0,001); 6 adolescents with high nutritional status and high risk of binge eating disorder; 97 adolescents with high nutritional status and low risk of binge eating disorder; 212 adolescents with normal nutritional status and low risk of binge eating disorder; 61 adolescents with high nutritional status and no risk of binge eating disorder; and 78 adolescent with normal nutritional and no risk of binge eating disorder. Conclusion: The binge eating disorder tendencies on adolescents with both high and normal nutritional status in Yogyakarta is significantly different.
Dr. SAID MOUSTAFA M,ELDEIB
1- Dr. Amr I. M. Hawal. Prime Health Care Group, Dubai - UAE. 2- Dr Said Moustafa M, Eldeib NMC Health Care Group, Abu Dahbi- UAE
Title: Necrotizing Enterocolitis in a Preterm Infant Newborn & Role of Feeding …. An Update!
Time : 10:00 AM
Biography:
Dr. Said is a Pediatrician and Neonatologist whose experience in the field spans 15 years, backed by a higher education degree from royal college of pediatrics’and child health in UK,in addition to master degree from Ain Shams university in Egypt one of the oldest and top ranking universities in the MENA region. He is pioneering an open and contextual evaluation model based on constructive responses, which has led in the creation of new methods to improve pediatric healthcare, neonatology and pediatric nutrition. Dr. said has established this model following his years of experiences in medical practice, research and evaluation, and teaching and administration in hospitals and medical universities in the reigion, including Egypt, kuwait and the UAE. Dr. said has
published studies in reputable international journals in neonatology and pediatric nutrition. He has also presented his findings in prestigious international conferences and symposia.
Abstract:
Statement of the Problem:
It’s a clinical case presentation of a male Preterm infant Newborn (+31 wks) who was delivered in our hospital & transferred to our NICU because of Prematurity, VLBW & need to respiratory support. Baby shortly undergo Necrotizing Enterocolitis (NEC) on 5th day of life shortly after start of expressed milk feeding …! Which was early detected by use of Near Infrared Abdominal spectroscopy (NIRS). Baby was deteriorated clinically in a couple of hours & undergo intestinal perforation with peritonitis , So, Abdominal exploration surgery with intestinal resection & end – to end anastomosis was done urgently.
Baby improved gradually & early feedings was started & gradually increased up to full feedings with use of Human Fortified Milk (HMF) & probiotics , Prebiotics.
Findings: The Study stated the evidence-based Feeding Strategies guidelines for necrotizing enterocolitis (NEC) among very low birth weight infants & Role of trophic feedings, Probiotics, Prebiotics & micronutrients in Prophylaxis, Prevention & Management of NEC.
Recommendations: 1) -Prematurity is the single greatest risk factor for NEC & avoidance of premature birth is the best way to prevent NEC. 2)-The role of feeding in the pathogenesis of NEC is uncertain, but it seems prudent to use breast milk (when available) and advance feedings slowly and cautiously. 3)-NEC is one of the leading causes of mortality, and the most common reason for emergent GI surgery in newborns. 4)-NEC remains a major unsolved medical challenge, for which no specific therapy exists, and its pathogenesis remains controversial. 5)-A better understanding of the pathophysiology will offer new and innovative therapeutic approaches, and future studies should be focused on the roles of the epithelial barrier, innate immunity, and microbiota in this disorder. 6)-Bioinformatics modeling is a new emerging strategy aimed at understanding the dynamics of various inflammatory markers and their application in early diagnosis and treatment.
K. M. Yacob (Chief Physician).
K. M. Yacob (Chief Physician). Marma Heatth Centre,Kochi ,Kerala,India
Title: Why W neurons decreases and C neurons increases in fever?
Time : 11:00 AM
Biography:
Abstract:
As you aware, if temperature increases (Absence of fever)after 31 degree Celsius , Warm sensitive neurons increase their firing rate and inhibit Cold sensitive neurons as core temperature increases. As temperature drops, the firing rate of Warm sensitive neurons decreases, reducing their inhibition, and Cold sensitive neurons which respond by increasing their firing rates.
On the contrary to increase of temperature, in fever the firing rate of Warm sensitive neurons decreases, the firing rate of Cold sensitive neurons increases as core temperature increases. inhibit warm sensitive neurons. The temperature increasing and decreasing controlled by the brain. The firing rate of Warm sensitive neurons and Cold sensitive neurons also controlled by the brain.
When the disease becomes threat to life or organs, blood circulation decreases. Temperature of fever will emerges to increase prevailing essential blood circulation.
WBC and their products stimulate the brain to increase temperature by increasing the firing rate of Cold sensitive neurons and decreasing the firing rate of Warm sensitive neurons. And it acts as a protective covering of the body to sustain life.
There is no way other than this for a sensible and discreet brain to increase temperature.
If the aim of Cold sensitive neurons increasing their firing rates in hypothermia is to increase temperature, then the aim of Cold sensitive neurons increasing their firing rates during fever is also to increase temperature.
How can we prove that W neurons decreases and C neurons increases in fever to protect the life or organ?
If we ask any type of question related to fever by assuming that the Warm sensitive neurons decreases and Cold neurons increases in fever to protect the life or organ we will get a clear answer. If avoid or evade from this definition we will never get proper answer to even a single question
If we do any type of treatment by assuming that the Warm sensitive neurons decreases and Cold neurons increases in fever to protect the life or organ , the body will accept, at the same time body will resist whatever treatment to decrease temperature and blood circulation.
No further evidence is required to prove The Warm sensitive neurons decreases and Cold neurons increases in fever to protect the life or organ.
Dr Lamidi Audu
XAudu LI1, Otuneye AT1, Mairami AB1, Mukhtar-Yola M1, Mshelia LJ1, Ekhaguere OA2 1. Paediatrics department, National Hospital Abuja, Nigeria. 2. Paediatrics department, Indiana University, Indianapolis, USA
Title: Gestational Age (GA) related Neonatal Survival at a tertiary health institution in Nigeria: the age of fetal viability dilemma.
Time : 12:00
Biography:
Abstract:
Background Although official age of fetal viability in Nigeria is 28 weeks, there are pockets of reports of survival of babies delivered at younger GA from different parts of the country. This is likely to generate important ethical and medical concerns in our approach to management of births occurring before the official GA of 28 weeks.
Aim/Objective: to determine the gestational age specific neonatal mortality among preterm deliveries at the National Hospital Abuja
Methods.A retrospective review of relevant data from the National Hospital Neonatal Registry Database (REDCap) was undertaken to determine the mortality rate of preterm babies managed in NICU from January 2017 to February 2018. Disaggregated GA specific mortality rates were also computed to determine the fetal age at which extra uterine neonatal survival rate was at least 50%. GA estimation was based on mothers’ LMP in over 96% of cases.
Results and conclusion; Sixty two of 305 preterm babies admitted died during hospitalization giving a mortality rate of 20.3%. This was significantly higher than the mortality rate among term babies (7.5%, P=0.01) hospitalized over the same period. Antenatal steroid use was low (11.2 %), 188(25.8%) received CPAP for RDS and none of the babies received surfactant.
There were no survivors among babies delivered at GA of 22-25 weeks (11, 3.6%). However survival rate at 26 weeks gestation was 53.8% and this subsequently increased reaching a peak of 80% survival at 36 weeks. RDS accounted for 53.9% of all deaths. GA, place of delivery and birth weight were significantly associated with risk of death.
It is concluded that the survival rate (53.8%) of babies at GA 26/52 despite minimal antenatal interventions and limited postnatal respiratory support was reasonably high and this could serve the basis for discussions for a downward review of age of fetal viability in Nigeria.